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Pugh, D

This is the compound's most clinically robust finding

Though, postmortem breakdown of GSH complicates interpretation of such findings 7

ArticlePubMed 14

Given the NCOA4 abundance is dually modulated by autophagy and the ubiquitin-proteasome system, its downregulation might indicate a feedback mechanism involving ubiquitin-dependent proteasomal degradation, which is promoted by high iron levels and involves iron-dependent binding to the E3 ubiquitin ligase HERC2 (137)