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To demonstrate its relevance in vivo , mice were administrated with AAV8-shRNA-LRP1 (KD) and AAV8-scramble-LRP1 (scr) intravenously, accompanied by CCl 4 -induced liver fibrosis (Fig

[103] incorporated covalent i , i + 4 lactam linkages to constrain the peptides derived from the p21 into the 3 10 -helical structure required for proliferating cell nuclear antigen (PCNA) inhibiting

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