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In accordance with theory of hormesis, we suppose that such mild, repeated stresses are responsible for adaptation of rats to subsequent stresses, e.g

However, it showed be noted that in animal models, there is a vast diversity of markers, both in serum (lipid profile, glucose, redox, etc.) as well as in tissues (redox activitySOD, catalase, glutathione peroxidaseGPx, MDA, carbonylated proteins, AGEs, 4-Hydroxynonenal4-HNE, among othersinflammatoryTNF-, IL-1, IL-6, etc.) or gene expression markers (proteins involved in pro or anti-inflammatory activity, glucose, and lipid metabolism, etc.), in addition to mitochondrial redox markers (8-OHdG, mitochondrial membrane potential, NADPH activity) that have been studied

GPX5 from C

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