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Activation of IGF-1 and insulin signaling pathways ameliorate mitochondrial function and energy metabolism in Huntingtons Disease human lymphoblasts

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To reach this goal, we used one ER + and two TNBC cell lines

Data availability The datasets presented in this study can be found in online repositories

This is important for further understanding functional redox relays in specific subcellular compartments ( Pf Prx1a is a cytosolic protein)

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