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Because the strategy lacks platelet-specific functions such as clot retraction and amplification mechanisms driven by platelet secretion, their ability to sustain long-term haemostasis is inherently incomplete, as reflected by occasional rebleeding in the tail-laceration assay (Supplementary Fig

Finally, we explore the therapeutic strategy of targeting this novel form of cell death, aiming to provide some insights for the management of atherosclerosis

Conclusion HAGSH scaffolds represent an intelligent, bioadaptive platform for regenerative wound therapy

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Liu H, Guo H, Deng H et al (2020a) Copper induces hepatic inflammatory responses by activation of MAPKs and NF-B signalling pathways in the mouse

Oxygen radicals in brain injury