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In sum, the evidence strongly supports membrane perturbation and altered phospholipid packing in mediating the electrophysiological and inotropic effects of LCACs in the heart

A previous study 286 revealed not only that p53 can modulate liver fibrosis by altering CTGF expression but also that the p53/CTGF pathway may be a new target in the treatment of liver fibrosis

They are characterized by chronic inflammation, and a dysfunctional immune system, involving joints, serosal membranes, connective tissue and blood vessels in multiple organs (1)

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