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In this review, we discuss the pipeline of obesity pharmacotherapies with focus on entero-pancreatic hormone-based molecules, and we evaluate the data from early and late phase clinical trials on their safety and efficacy on WL in people with and without T2D

This has not been demonstrated in animal studies, but the mechanism warrants caution

References 1 Proc Natl Acad Sci USA (1977)Schoenenberger GA, Monnier M Characterization of a delta-electroencephalogram (-sleep)-inducing peptide isolated from rabbit brain

The CagriSema metabolic preservation mechanism, specifically the maintenance of mitochondrial function in skeletal muscle during weight loss, could theoretically slow the rate of weight regain by preventing some of the metabolic adaptation that makes regain almost inevitable