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Some mature osteoblasts are, subsequently, embedded in the matrix as osteocytes, which express the dentin matrix protein-1 (DMP-1)

I269T (a common missense variant of NPT1) mutations increase the maximum volume by increasing the turnover rate of the urate transport and output tomitigate gouty risk, but do not change NPT1 membrane expressions (Sakiyama et al., 2016)

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Activation of GHS-R1a by these ligands has been associated with differential intracellular calcium responses and variable engagement of downstream effector pathways in cell-based studies of mammalian research models

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