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Given the consequences of genotoxic stress, counteracting DNA damage in the body may take the form of firstly, DNA repair mechanisms being enhanced, or secondly, preventing DNA damage through strengthening the integrity of the epidermal barrier between UVR and our UV-labile molecular components

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Mutations in EGFR affect TME components, including myeloid-derived suppressor cells (MDSCs), TILs, tumor-associated macrophages (TAMs), immunoregulatory cytokines, and regulatory T cells (Tregs) [59]

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