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A diagram illustrating the study development on the epidemiology and mechanism of DMED is presented in Figure 1

Gapdh was used as an internal control

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Elimination half-lives were similar between the disease groups (range 1.11.5 h), whereas T max was delayed by 1 h in NAFLD patients, suggesting that a reduced silymarin metabolism may cause the differences in drug exposure between NAFLD and HCV subjects [32]