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By attenuating oxidative damage and modulating inflammatory factors and fibrotic cytokines (eg, I/III Collagen type 1, intercellular adhesive molecule-1, fibronectin, inducible nitric oxide synthase, and arginase type 1) during disease progression, Sal B-DPI significantly alleviated bleomycin-induced pulmonary fibrosis as evidenced by the reduction of alveolar wall congestion, inflammatory cell infiltration, emphysema extent, and lung ventilatory function

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