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It promotes the well-being of your blood vessels by safeguarding them from inflammation and oxidative damage

During this process, instead of ADH, cytochrome P450 2E1 (CYP2E1) is activated in the liver to break down acetaldehyde [6]

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These findings suggest that ferritinophagy-derived iron and TFRC-mediated endolysosomal iron trafficking may converge within lysosomes to create a vulnerable pool of catalytic Fe 2+ able to initiate ferroptotic damage when properly engaged, thereby linking iron storage turnover, stem-like phenotypes, and ferroptosis sensitivity [36]