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Simultaneously, silybin improves endothelial dysfunctiona critical link in the onset of inflammationby lowering asymmetric dimethylarginine (ADMA) levels, thereby reversing nitric oxide synthase inhibition and increasing the bioavailability of NO, which possesses anti-inflammatory and vasodilatory functions

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Many groups, including ours, have addressed molecular alterations in peripheral tissues in association with symptomatic changes in major illnesses

The GIP component becomes more active at this dose, affecting lipid metabolism

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