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This is particularly relevant when we consider GSH pathways, as well as their transcriptional regulator Nrf2, for proliferation, survival and function of T cells, B cells and macrophages (325, 326)

It has been suggested that an inadequate dietary intake of these trace minerals may compromise the effectiveness of these antioxidant defense mechanisms

However, as reiterated many times in this review, the mechanism of -synuclein fibrillation is as complex as it can get

doi: 10.1039/b9nr00178f

Formation of the NFT radical anion by cellular reductases To elucidate the mechanism of Nrf2 activation by NFT, we focused on the ability of NFT to yield an activated species capable of oxidizing critical sulfhydryl groups in the Nrf2-Keap1 complex

Animal studies demonstrate that dihydrotestosterone (DHT) treatment upregulates ferroptosis-related markers ( e.g ., ACSL4) in ovarian cells, suggesting that androgens may exacerbate ferroptosis by modulating lipid metabolism genes (43)