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In particular, PDAC cells require Gln, and supplementation of aKG with nonessential amino acids (NEAAs) rescues PDAC cells from Gln deprivation-mediated growth inhibition 11,26 , supporting the present data showing that Gln is a critical source for aKG production

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doi: 10.3390/ijms22052734 32 YanHWangLZhangGLiNZhaoYLiuJet al

Mutation-independent therapies can have various downstream targets that aim to improve mitochondrial respiration, reduce mitochondrial stress, inhibit or delay RGC apoptosis, and/or promote RGC survival