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Although TfR-mediated transcytosis and related strategies can improve CNS delivery, clinical deploymentparticularly in the setting of anti-angiogenic-therapy-induced BBB remodelingmust balance molecular size (tissue penetration), half-life, and effector function

Activated AKT1 phosphorylates PRMT7 at T73 residue thereby increasing the GLUD1 stabilization through PRMT7-mediated methylation and inhibiting GLUD1 ubiquitin-dependent degradation, which ultimately facilitates gastric cancer cell proliferation and migration

MET15 / met15 0

Sarangi, P.K

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Constitutional BRCA1 methylation and risk of incident triple-negative breast cancer and high-grade serous ovarian cancer