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More intriguingly, the cross-link between ferroptosis and inflammation is mutually induced and drives a local autoamplification loop[60]

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Since BT474 and BT474-R tumour cells display distinct growth rates, we tested different bioprinting concentrations of parental or resistant cells, and followed their proliferation in time by regular Live/Dead staining

When combined, ferroptotic lipid peroxidation and MQC failure seem to be proximal upstream factors that establish the Nrf2-NLRP3 rheostat in PD

NFAT is positively regulated by IL-1, and in a positive feedback loop, IL-1 expression is dependent on NFAT and L-type Ca 2+ channels (169)

Marcel Dekker, Inc, 381-399, 1990