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Description

The development of the obesity phenotype constitutes dramatic alterations in adipocytes, along with several other cellular mechanisms which causes substantial increase in systemic oxidative stress mediated by reactive oxygen species (ROS)

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The PrxIII KO mice were observed to have increased levels of mitochondrial ROS generation, adipogenic markers, aP2, CCAAT/enhancer binding proteins (C/EBP), and peroxisomal proliferator activated receptor (PPAR), and protein carbonylation