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BMC Microbiol 25:250

10.1134/S0006297922120112 31 HajoschR.SuckfuellM.OesserS.AhlersM.FlechsenharK.SchlosshauerB

Its action on these three receptors suggests a complementary mechanism where glucagon-induced gluconeogenesis is inhibited by GLP-1/GIP, and glucagons enhancement of energy expenditure is augmented by the appetite-suppressing effects of GLP-1/GIP, thereby promoting weight loss [112]