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MTAP deficiency prevents tumors from recycling the polyamine metabolism byproduct methylthioadenosine (MTA) back into Met, forcing reliance on exogenous Met, while simultaneously increasing sensitivity to purine synthesis inhibitors (68)

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their mean age was around nine years old, and all of them were on HU stable dose at enrollment, as long-term use of HU is safe and well-tolerated in large cohorts of children and adults with SCD, reducing mortality and morbidity of both children and adult patients [30, 31]

Higuchi, "Ubiquinol-10 Supplementation Activates Mitochondria Functions to Decelerate Senescence in Senescence-Accelerated Mice", Antioxid

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