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The primary antioxidant defense in cells is mediated through enzymatic and non-enzymatic systems, including factors such as glutathione (GSH), glutathione peroxidase (GPX), catalase (CAT), and superoxide dismutase (SOD) (Mourente et al., 2002)

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Effect of glutathione depletion on Glutathione S-Transferase and Glutathione reductase activities In view of the major changes induced DEM but not BSO on cell proliferation we determine GST and GR activity in 3T3 fibroblasts (table 1 and table 2, respectively, supplementary data)

Advanced oxidation protein products (AOPPs) were identified in the plasma of uremic patients by Witko-Sarat et al

Mitophagy decline in aging hearts [12] and impaired PINK1/Parkin-mediated mitophagy in AD [13, 14] lead to damaged mitochondrial accumulation

This combined assault ultimately disrupts neuronal homeostasis, contributing to the structural and functional impairments underlying the observed cognitive and neuropsychiatric sequelae