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During the resolution of inflammation, SPMs produced by polymorphonuclear cells and macrophages act to limit local tissue damage caused by inflammatory responses, including signaling cessation of polymorphonuclear cell infiltration, macrophage switching to an anti-inflammatory phenotype, apoptotic cell clearance, and reducing pain caused by nociceptor sensitization (49, 61)

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Antigen Background The glutathione S-transferases (GSTs) are a multigene family of isoenzymes which catalyze the conjugation of glutathione to electrophilic substrates