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[DOI] [PubMed] [Google Scholar] 82.Tajik N., Frech M., Schulz O., Schlter F., Lucas S., Azizov V., Drholz K., Steffen F., Omata Y., Rings A., et al

Wiig H, Reed RK, Tenstad O

10.1073/pnas.1812405116 Proc

Here, we analyzed CNV lesion numbers and sizes in a large number of agematched (6weekold) mice and could show that targeting Nlrp3 or Il1r1 significantly reduced CNV lesion numbers (but not CNV lesion sizes), while targeting Il18 did not show a statistically significant change in either CNV lesion numbers or their size (Fig 8A and B, and Appendix Fig S5A and B)

Next, the study showed that inhibition of Sat1 alleviated MIRI by suppressing ferroptosis in vivo and in vitro , and over-expression of Sat1 promoted MIRI via activation of ferroptosis