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MSCs exert their therapeutic effects on PNI possibly through their dual functions, multipotent differentiation capacity into neural type of cells and their paracrine secretion of an array of bioactive factors with multiple trophic effects on both neural and nonneural cells [13], whereby the potent immunomodulatory effects of MSCs on immune cells, particularly macrophages, may constitute one of the major mechanisms by which MSCs exert their therapeutic effects on PNI [14]

Furthermore, during cellular uptake and metabolic absorption, EVs themselves degrade quickly or are actively cleared by the body, leading to a high degradation rate (Tran et al., 2020)

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The mechanisms underlying GHK-Cu's anti-inflammatory effects involve multiple molecular pathways that converge on the resolution of inflammation

Disorders of collagen biosynthesis and structure

Xiang et al., 2006) and the small Rho-like GTPase TC10 (Mayer et al., 2013) that in turn regulate cluster formation and the aggregation GABA A Rs (Poulopoulos et al., 2009