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This upregulation of miR-449a inhibits the expression of its target gene ACSL4, thereby suppressing neuronal ferroptosis (139)

This rhythm originates from the suprachiasmatic nucleus (SCN), where light exposure transmits neural impulses to the SCN via the retinohypothalamic tract, synchronizing the activity of the SCN and the nocturnal secretion rhythm of melatonin with the 24-h light/dark cycle

If therapy is unsuccessful, the brain loses as many neurons each hour as it does in approximately 3.6 years of average aging [16]

This is difficult due to the cost, as well as disagreement between experts on evaluation and remediation

For first-line drugs, isoniazid (INH) resistance is primarily associated with mutations in the katG gene, which encodes the catalase-peroxidase enzyme responsible for activating INH (Rivire et al., 2020)