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Unraveling mucin domains in cancer and metastasis: When protectors become predators

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As such, H3.3K27M mutation accompanied by increased total H3K27ac and reduction in H3K27me3, leading to glioma formation and subsequent tumor progression in a brainstem glioma model 139

and microglial involvement and transition toward disease-associated cell states were suggested (Maniatis et al., 2019

Furthermore, it enhances osteogenesis in calvarial defects of T1DM rats, suggesting a novel therapeutic strategy for diabetes-associated impairment of bone regeneration

These use considerations are presented alongside the label guidance in Table 2