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Consistently, the motor nerve conduction study confirmed the absence of demyelinated processes in the tail caudal nerves after spinal cord injury (the CMAP showed normal biphasic potentials, similar amplitudes, and similar conduction velocities in all of the rats) (Table 4)

The peptide counteracted picrotoxin (non-competitive GABA receptor Cl channel blocker), isoniazid (inhibitor of glutamic acid decarboxylase and GABA synthesis), and bicuculline (GABA-A antagonist), all through dose-dependent protective effects without producing behavioral changes when administered alone