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Abstract Diabetic kidney disease, known as a glomerular disease, arises from a metabolic disorder impairing renal cell function

Chen, Morphological evolution for CeO2 nanoparticles synthesized by precipitation technique

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demonstrated in enteric glial cell models that PS-NPs accelerate the aggregation of mutant A53T -synuclein, disrupt mitochondrial membrane integrity, and impair lysosomal function, collectively exacerbating dysregulation of the mitochondrial-lysosomal axis 41

(2) Under the action of catalytic residues, the N-acetylglucosamine (GlcNAc) carbonyl oxygen of the substrate itself acts as an intramolecular nucleophile, attacking the heterologous carbon (C1) of the same sugar ring, forming a covalent oxonium ion intermediate accompanied by C1 configuration inversion, and completing the cleavage of the non-reducing side glycosidic bond of the glycosidic oxygen atom