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STAT5b in human subjects is critical for GH-induced IGF-I expression and growth promotion as demonstrated by the identification of mutations in the STAT5b gene of patients presenting with severe GH insensitivity in the presence of normal GHR (58)

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We then examined the binding interface between DET1 and Ube2e2

Indeed, APR-246 was synergistic with GPX4 inhibitor, ML-210, in parental OVCAR5 and SKOV3 (Fig