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This pathway has been studied in several in vitro models and is under investigation as a potential therapeutic target in several cancers, including breast, ovarian, liver, and prostate 15,16,17,23

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So, the model was transposed into a quasiconvex function by returning the highest negative glutathione flux and report enzyme level at the point

Cancer cells deploy lipocalin-2 to collect limiting iron in leptomeningeal metastasis

CHAC1 degradation of glutathione enhances cystine-starvation-induced necroptosis and ferroptosis in human triple negative breast cancer cells via the GCN2-eIF2-ATF4 pathway

hormone production and secretion) or pathological (i.e