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Diabetes is the leading risk factor for severe illness, with in excess of 40% of melioidosis sufferers having diabetes, or being diagnosed with diabetes at the time of hospital presentation [Citation6]

Strober W, Asano N, Fuss I et al (2014) Cellular and molecular mechanisms underlying NOD2 risk-associated polymorphisms in Crohns disease

Erythropoietin synthesis in renal myofibroblasts is restored by activation of hypoxia signaling

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This "gene reset" capability is one of the most exciting areas of GHK-Cu research