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This could be explained either by the simulated anthropogenic variability being too low, as suggested by Casado et al

diabetes obesity, non-alcoholic fatty liver disease, non-alcoholic steatohepatitis (NASH), a cardiovascular disease or any other disease/disorder involving GLP-1 (including any disease/disorder involving, or mediated by, a deficiency of GLP-1 or deficient GLP-1 signalling), using the solid oral pharmaceutical composition provided herein

ICH harmonised guideline: S2(R1) Genotoxicity testing and data interpretation for pharmaceuticals intended for human use (Current Step 4 version)

This suggests that non-responders were less severely affected by ME/CFS at baseline, and that the main positive effect of nasal B12 administration occurs in more severely affected patients

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