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Description

The fosgonimeton clinical history is not direct evidence about Dihexa, but it is relevant context for any reader evaluating the HGF/c-Met pathway as a research target for cognitive applications

10.1038/s41583-024-00813-1 22 BhuiaM

AOD9604 represents part of a broader scientific effort to develop targeted therapies that can isolate beneficial effects of growth hormone while avoiding unwanted systemic effects, similar to the development of selective androgen receptor modulators (SARMs) or selective estrogen receptor modulators (SERMs) in other therapeutic areas

It acts by binding to BCL-W, BCL-2, and BCL-XL, inhibiting their function [102]

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The hormone promotes gluconeogenesis (glucose production) in the liver and reduces glucose uptake in peripheral tissues, creating an insulin-resistant state