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Two possible sites were identified in the catalytic subunit of glutamate-cysteine ligase (GCLC), the rate limiting enzyme for GSH synthesis

However, researchers assume that patients with type 2 diabetes mellitus can no longer metabolize fatty acids optimally through the mitochondria

This excess lactate contributes to an acidic tumor microenvironment (TME) that facilitates cancer cell invasion, metastasis, and immune evasion by suppressing cytotoxic T cells and natural killer (NK) cells [85,86,87]

10.1371/journal.pone.0037971 PLoS One 19 BindelsL.NeyrinckA.ClausS.Le RoyC.GrangetteC.PotB.et al (2016)

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