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136 In a UC rat model, pretreatment of bone marrow MSCs with IL-6 increased their ability to inhibit inflammatory factors

These observations were confirmed in LIN28A-depleted hPSCs, suggesting that the depletion of LIN28A significantly upregulated UPF1-UPF2 interactions (Fig

Under pathological conditions, liver macrophages can polarize into distinct phenotypes (M1 and M2), which play crucial roles in promoting inflammation and liver fibrosis