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Demedts M, Behr J, Buhl R, et al

Relevantly, familial PD is mainly characterized by mutations in genes involved in mitochondrial dysfunction, such as Parkin, -synuclein, and leucine-rich repeat kinase 2 (LRRK2) [103,105,106,107]

Transcriptomics in toxicology

However, it has been found that greater ROS levels that push cancer cells beyond the breaking point lead to the activation of different cell death mechanisms, indicating the tumor-suppressing perspectives [7]