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T cells that are bereft of operational leptin receptors display a hindered ability to morph into Th17 cells, an outcome stemming from subdued activation of STAT3 and its associated signaling trajectories (87)

C.JeongE.KimE.HongS

Evidence has been documented by postmortem studies pointing to the increased apoptotic activity in autistic individuals

Our earlier studies have shown that treatment of HT22 cells with glutamate activates PDI-mediated NOS dimerization, which is followed by accumulation of cellular NO and ROS, and ultimately, oxidative cell death [8]