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100 Therefore, in addition to tumorous glial cells, these microenvironmental macrophages are also potential therapeutic targets for neurofibroma and glioma

For example, in conditions such as inborn errors of metabolism (e.g., medium-chain acyl-CoA dehydrogenase deficiency), accumulating acyl-CoA intermediates are converted into acylcarnitines and excreted via urine

The mGluR5 antagonist 2-methyl-6-(phenylethynyl) pyridine reduced the expression of CREB, p-ERK, and Rad51 in the rotenone-induced PD rat model and alleviated the DNA damage of dopaminergic neurons [183]

Detoxification : The liver relies on glutathione to detoxify harmful substances, including heavy metals and environmental pollutants

Vagnozzi R, Signoretti S, Cristofori L et al (2010) Assessment of metabolic brain damage and recovery following mild traumatic brain injury: a multicentre, proton magnetic resonance spectroscopic study in concussed patients

Pivotal advance: Toll-like receptor regulation of scavenger receptor-a-Mediated phagocytosis