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No hGH Receptor Binding or IGF-1 Stimulation AOD9604 did not induce hyperglycaemia or reduce insulin secretion in sharp contrast to chronic intact hGH treatment and confirmed that the concept of hGH behaving as a pro-hormone is valid, with fragments capable of acting via mechanisms novel to traditional GH receptor-stimulated pathways

This designation allows the use of research chemicals strictly for in vitro testing and laboratory experimentation only

Public written comments can be submitted to docket FDA-2025-N-6895 through July 22, 2026

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Inhibition of NNMT has been explored in preclinical research for potential effects on adiposity regulation, insulin sensitivity, and metabolic efficiency