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Sarrah serves as an anti-apoptotic lncRNA in cardiomyocytes after acute myocardial infarction (AMI) and was demonstrated to be downregulated in aging [168]

This phenomenon is also known to prevail in RA that displays a rapid proliferation rate and strong invasion ability of synovial fibroblasts [18], increased local energy demands due to pannus formation, and hypoxia caused by the dysregulated microvasculature [19]