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It regulates numerous genes associated with ferroptosis, including those involved in GSH synthesis, NADPH regeneration, and iron metabolism (such as iron storage and export, as well as heme synthesis and degradation)

Moreover, excess iron could promote the formation of reactive oxygen species (ROS), which can lead to oxidative stress and DNA damage, with cytokine-regulated inflammatory pathways and a central contribution from innate immune cells in the tumor microenvironment (360, 361)

6.2 Innovation in clinical trial design Traditional PD trials are constrained by: Slow progression of disease markers

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