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Although the direct cytotoxic effects of high cellular GSSG levels have been documented in a variety of cell types 33,34,35,36 , our cell culture studies with hepatocytes revealed that increased GSSG accumulation, despite being non-toxic per se, could sensitize hepatocytes to TNF-induced killing, suggesting that GSSG and TNF can synergistically induce hepatocyte death and that GSSG-triggered sensitization to TNF hepatotoxicity represents a clinically relevant process that may account for disease progression in NAFLD

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Ameliorative effects of histidine on oxidative stress, tumor necrosis factor alpha (Tnf-Alpha), and renal histological alterations in streptozotocin/nicotinamide-induced type 2 diabetic rats

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Of note, TCE-mediated skewing of hepatic and splenic immune lineage development was effectively reversed by NAC (Table 1)

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