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Oxidative stress releases pro-inflammatory molecules that interact with ROS to produce inflammation, performing major role in early onset of aging and metabolic diseases [7]

Its primary job is to neutralise free radicals, which are unstable molecules that can cause oxidative stress and damage to our cells

and CAT against H 2 O 2 and GPx, which catalyzes the reduction of H 2 O 2 or organic hydroperoxides to water or corresponding alcohols by glutathione GSH, thus playing a central role in the mammalian antioxidant system [29]

Conditions that are related to mitochondrial dysfunction and reliant on FAD and FMN include Parkinsons disease, Alzheimers, migraines, Complex I deficiency, Alpers syndrome, multiple sclerosis, and Kearns Sayre syndrome.[ref] Nutrient interactions: Riboflavin also helps convert tryptophan to niacin (vitamin B3), which in turn activates vitamin B6 .[ref] It is also essential for the methylation cycle and folate-related metabolism

Environmental factorstemperature, light exposure, and moistureare relentless adversaries in the battle for peptide stability

Studies give extremely different results some show that high levels of IGF-1 in the elderly increase the risk of dementia, other that low levels of IGF-1 increase this risk