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PubMed Jastreboff, A.M., Kaplan, L.M., Fras, J.P., Wu, Q., Du, Y., Gurbuz, S., et al

In a study on HCC cell lines and mouse models with in situ tumors, it was shown that MET activation can phosphorylate the 56th tyrosine residue and trigger glycogen synthase kinase 3 (GSK3), resulting in decreased PD-L1 expression [137]

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It was shown that the toxicity of OTA could be decreased by maintaining glutathione production with N -acetyl cysteine (NAC), which decreased reactive oxygen species (ROS) and 8-oxoguanine formation