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Impaired extracellular secretion of mutant superoxide dismutase 1 associates with neurotoxicity in familial amyotrophic lateral sclerosis

This shift is highly indicative that their engagement with the DBCO landmark is greatly improved upon the removal of the physical barrier (e.g., OM and cytosolic membrane)

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IL36RN mutations affect protein expression and function: a basis for genotypephenotype correlation in pustular diseases

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