[DOI] [PubMed] [Google Scholar] 3.Bavarsad Shahripour R., Harrigan M.R., Alexandrov A.V
Perrotta et al., 2020), altering nitrogen and phosphorus availability (Swamikannu and Hoagland, 1989
Hemopexin Therapy Reverts Heme-Induced Proinflammatory Phenotypic Switching of Macrophages in a Mouse Model of Sickle Cell Disease
7-DR2 (7-Dehydrocholesterol reductase), AGL6 (Agamous-like 6), AGO7 (Argonaute7), ALC (Alcobaca), ALD1 (AGD2-like defense response protein), ALMT (Al-activated malate transporter),ALS1 (Acetolactate synthase 1), AMS (Aborted microspores), AN2 (Anthocyanin2), ANT1 (Anthocyanin 1), AP2a (Apetala2a TF), APX2/4 (Ascorbate peroxidase2/4), Blc (Beta-lycopene cyclase),BOPs (Blade-on-petiole), BZR1 (Brassinazole-resistant 1), CRTISO (Carotenoid isomerise), CAT9 (Cationic amino acid transporter 9), CBF1 (C-repeat/dehydration responsive element binding factor1), CLV3 (Clavata3), CMT4 (Chromomethylase), CNR (SBP-box colorless non-ripening), CPK28 (Calcium-dependent protein kinase28), CP (Coat protein), CrtR-b2 (Beta-carotene hydroxylase 2),CRTISO (Central role of carotenoid isomerase), CYC-B/CycB (Lycopene beta cyclase), DDB1 (DNA damage UV binding protein 1), DELLA (Aspartic acidglutamic acidleucineleucinealanine),DCL2b (Dicer-like 2b), DET1 (Deetiolated1), DMR6 (Downy mildew resistance 6), EJ2 (Enhancer-of-jointless2), ENO (Excessive number of floral organs), EPSPS (5-Enolpyruvylshikimate-3-phosphate synthase), ETR1 (Ethylene receptor 1), FASCIATED (FAS), FMO1 (Flavin-dependent monooxygenase), FUL1/2 (Fruitfull), GABA-TP1 (Pyruvate-dependent g-aminobutyric acidtransaminase 1), GAD2 (Glutamate decarboxylase 2), GAD3 (Glutamate decarboxylase 3), GF (Greenflesh/Staygreen), GGP1 (GDP-l-galactose phosphorylase1), GRXS (CGFS-type glutaredoxin),GSTAA (Glutathione S-transferase), HAK20 (High-affinity K+ 20), HKT1;2 (High-affinity potassium transporter 1;2), HY5 (elongated hypocotyl5), HyPRP1 (Hybrid proline-rich protein 1), IAA9(Auxin-induced 9), INVINH1 (Invertase inhibitor 1), J2 (Jointless-2), JAZ2 (Jasmonate zim domain), LBD40 (Lateral organ boundaries domain40), LOCULE NUMBER (LC), LCY-B1 (Lycopene bcyclase1), LCY-B2 (Lycopene b-cyclase 2), LCY-E (Lycopene e-cyclase), LIN (Long inflorescence), MAPK3 (Mitogen activated protein kinase 3), MIR164A (MicroRNA164A), TFAM1/TFAM2(Mitochondrial transcription factor A), Mlo1 (Mildew resistance locus o 1), MS10 (Male sterile 10), Ms1035 (Male sterile 1035), MULT (Multiflora), MYBS2 (MYB transcription factor S2), MYC2(Basic helixloophelix transcription factor), NAC (NAM-ATAF-CUC), NAM1/2/3 (No apical meristem1/2/3), NAC-NOR (NAC TF non-ripening), NOR (Non-ripening), NOR-like1 (Non-ripeninglike1), NPR1 (Nonexpressor of pathogenesis-related gene 1), O (ovate), ORRM4 (organelle RNA recognition motif-containing protein4), PDS (Phytoene desaturase), OFP (OVATE family protein),PG (Polygalacturonase), PG2a (polygalacturonase 2a), PHO1 (Phosphate 1), PIF4 (Phytochrome interacting factor 4), PL (pectate lyase), PMR4 (Powdery mildew resistance 4), PR-1 (Pathogenesisrelatedprotein-1), PRO (Procera), ProSys (Prosystemin), PSY1 (Phytoene synthase 1), RAD51/54 (DNA repair and recombination protein51/54), RBOH/RBOHE (Respiratory burst oxidasehomolog), RDR6 (RNA ald1dependent RNA polymerase 6), REP (Replicase), RIN (Ripening inhibitor), RMC (Reduced mycorrhizal colonization), SBPase (Sedoheptulose-1,7-bisphosphatase), S(Compound inflorescence), SCR (Scarecrow), SHR (Shortroot), SGR1 (Stay-green 1), SGS3 (Suppressor of gene silencing 3), SOS1 (Salt overly sensitive 1), SP (Self pruning), SP5G (Self pruning 5G),SSADH (Succinate semialdehyde dehydrogenase), Target-AID (Target activation-induced cytidine deaminase), TBG4 (b-galactanase), TFM6 (Tomato fruit malate on chromosome6), TMF(Terminating flower), ToMV (Tomato Mosaic virus), TRM3/4/5 (TONNEAU1 Recruiting Motif3/4/5), VPE5 (Vacuolar processing enzyme5), and WUS (Wuschel).RNPs (ribonucleoproteins), PEG (polyethylene glycol), Target-AID (Target Activation Induced Cytidine Deaminase), NHEJ (Homologous-End-Joining), KO (gene knock-out), KI (gene knock-in), HDR (homology-directed repair), HR (homologous recombination), HKI (HR-based KI)
(May 2006)
Rothberg et al., 2010) but has no overall impact on airway destruction and disease progression (Leibovitz, 2008
