glutathione after radiation treatment in Skin Aging and Tissue Regeneration: A Systematic Review of Molecular Mechanisms, Redox Modulation, and Biomedical Implications Tissue fibrosis induced by radiotherapy:

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In addition to treating diseases by modulating palmitoylation-related enzymes, therapeutic agents can target specific palmitoylated cysteine residues, such as C176/178, H-151, NO2-FAs or nitrofuran molecules, which can block STING palmitoylation modification by covalently modifying the Cys91 residue of STING, inhibiting type I interferon signaling, and providing a new therapeutic target for STING-associated diseases by blocking STING palmitoylation and inhibiting type I interferon signaling (103, 251)

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Hence, riboflavin supplementation in patients with GFER mutations may potentially have clinical benefit, but this is speculative

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