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MiR-33a and MiR-33b promote atherosclerosis by impairing cholesterol efflux and promoting inflammatory M1 macrophage conversion (78-80).Other microRNAs including MiR-223 and MiR-93 exhibit atheroprotective effects by increasing cholesterol efflux and conversion to the anti-inflammatory M2 macrophage phenotype (76,81-83)

However, these signals can travel with the circulatory system and reach different tissues, not only confined to the lungs

doi: 10.1007/s12035-022-03148-z 48

Synthesis, biological evaluation, and molecular docking study of novel allyl-retrochalcones as a new class of protein tyrosine phosphatase 1B inhibitors Bioorg

Oxidant formation can also arise via electron-leakage from mitochondria, from enzymes such as xanthine oxidase and lipoxygenases, and by uncoupling of enzymes such as endothelial and inducible nitric oxide synthases (NOS) (reviewed) 8