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Mechanistically, QYG treatment inhibited histone deacetylase expression and activity [96]

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Catechol estrogen metabolic pathway Strong evidence from studies of estrogen metabolism, formation of DNA adducts, mutagenicity, cell transformation and carcinogenicity led to and supports the hypothesis that specific estrogen metabolites, the catechol estrogen quinones, can react with DNA to form estrogen-DNA adducts in critical genes that lead to the initiation of cancer [11, 12]

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