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507 The brains of individuals with preclinical AD (PCAD) and AD exhibit oxidative damage to a variety of molecules, e.g., accumulation of protein carbonyls (PCs) in regions that are rich in A-peptide-containing SPs, increased lipid peroxidation in AD and PCAD hippocampi and elevated DNA damage

This convergence renders the NF-B and C/EBP pathways promising drug targets for the modulation of the SASP

[23] Most long-term effects are caused by damage to DNA

Importantly, integrating autophagy modulation with immunotherapy is emerging as a novel approach

Eur J Endocrinol (2015) 173:7919

PROMOTES PHYSICAL PERFOMANCE: Convert the fatty acids into energy to the cells and improve the symptoms such as fatigue and weakness